Pharma company: trinity chiesi pharmaceuticals
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List of trinity chiesi pharmaceuticals products:
- Acitak
- Adipine - Pharmacological action Adipine is aSelective calcium channel blocker class II, a derivative of dihydropyridine. This medication inhibits the entry of calcium into cardiomyocytes and vascular smooth muscle cells. Nifedipine has antianginal and hypotensive action. This drug lowers the tone of vascular smooth muscle. Adipine dilates coronary and peripheral arteries, lowers blood pressure and slightly - myocardial contractility, reduces afterload and myocardial oxygen demand. This medicine...
- Angeze
- Angitil
- Artracin
- Atimos Modulite
- Calfovit D3
- Cardioplen
- Centrapryl
- Curosurf
- Felicium
- Fenoket - Pharmacological action Ketoprofen is a NSAID, propionic acid derivative. This medication has analgesic, anti-inflammatory and antipyretic activity. The mechanism of action is associated with inhibition of COX activity - the main enzyme metabolism of arachidonic acid, a precursor of prostaglandins, which play a major role in the pathogenesis of inflammation, pain and fever. A pronounced analgesic effect of ketoprofen is due to two mechanisms: peripheral (indirect, via inhibition of...
- Fentamox
- Frador
- Glau-Opt
- Glucamet
- Hyteneze
- Infestat
- Ketocid - Pharmacological action Ketoprofen is a NSAID, propionic acid derivative. This medication has analgesic, anti-inflammatory and antipyretic activity. The mechanism of action is associated with inhibition of COX activity - the main enzyme metabolism of arachidonic acid, a precursor of prostaglandins, which play a major role in the pathogenesis of inflammation, pain and fever. A pronounced analgesic effect of ketoprofen is due to two mechanisms: peripheral (indirect, via inhibition of...
- Libetist
- Lofensaid
- Lopranol LA
- Maximet SR
- Meflam
- Monomax
- Muscinil
- Natramid
- Nimodrel - Pharmacological action Nifedipine is aSelective calcium channel blocker class II, a derivative of dihydropyridine. This medication inhibits the entry of calcium into cardiomyocytes and vascular smooth muscle cells. Nifedipine has antianginal and hypotensive action. This drug lowers the tone of vascular smooth muscle. Nifedipine dilates coronary and peripheral arteries, lowers blood pressure and slightly - myocardial contractility, reduces afterload and myocardial oxygen demand. This...
- Oprisine
- Optil
- Opumide
- Opustan
- Pralenal
- Probeta LA
- Pulvinal Beclometasone Dipropionate
- Pulvinal Salbutamol - Pharmacological action Albuterol Pharmaceuticals is a beta adrenoagonists with a predominant effect on beta2-adrenergic receptors (localized, particularly in the bronchi, myometrium, blood vessels). This medication prevents and reduces or eliminates bronchospasm, reduces the resistance in the airways, increases the vital capacity. It prevents the release of histamine, slow reacting substance from mast cells and factors chemotaxis of neutrophils. Compared with other drugs of this group has...
- Ventmax
- Vertab
- Virovir - Pharmacological action Acyclovir Pharma is an antiviral agent. Thymidine kinase of virus-infected cells actively converts acyclovir through a series of sequential reactions in the mono-, di- and triphosphate of acyclovir. The latter interacts with the viral DNA polymerase, and embedded in DNA, which is synthesized for new viruses. Thus it is formed a "defective" viral DNA which leads to suppression of replication of new generations of viruses. This mediciine acyclovir is active against...
- Viz-On
- Volsaid
- Zida-Co
- Zileze - Pharmacological action Zopiclone is a sleeping drug from group of cyclopyrrolone derivatives. This drug is "non benzodiazepine" agonist of benzodiazepine receptors. Zopiclone also has sedative, anxiolytic, muscle-relaxing center, anticonvulsant and amnesic properties. Like a derivative of benzodiazepine zopiclone enhances GABA-ergic processes in the brain, interacting with the benzodiazepine receptors, as a result of the increased sensitivity of GABA-receptors to the neurotransmitter....
- Zoxin
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The information was verified by Dr. Rachana Salvi, MD Pharmacology
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